RACIAL DIFFERENCES IN VASCULAR FUNCTION FOLLOWING INDUCED ACUTE INFLAMMATION

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dc.contributor.advisorRanadive, Sushant Men_US
dc.contributor.authorChesney, Catalina Anneen_US
dc.contributor.departmentKinesiologyen_US
dc.contributor.publisherDigital Repository at the University of Marylanden_US
dc.contributor.publisherUniversity of Maryland (College Park, Md.)en_US
dc.date.accessioned2020-10-08T05:32:41Z
dc.date.available2020-10-08T05:32:41Z
dc.date.issued2020en_US
dc.description.abstractAfrican-Americans (AAs) have higher rates of cardiovascular disease (CVD), including hypertension and stroke, as compared to their Caucasian-American (CA) counterparts. High resting concentrations of systemic inflammatory biomarkers contribute to vascular dysfunction and are predictive of future cardiovascular events; differential resting levels of inflammatory markers between groups may reveal increased potential for CVD in at-risk groups. Additionally, impaired endothelial function and increased arterial stiffness, subclinical measures of CVD progression, have been reported in AA groups. The purpose of this study was to examine race differences between young, healthy AA and CA adults after a systemic inflammatory stimulus and subsequent endothelial responses to inflammation. Endothelial function, arterial stiffness, and hemodynamic variables were measured. The results suggest there were no race differences in vascular function or hemodynamic responses following an acute inflammatory stimulus.en_US
dc.identifierhttps://doi.org/10.13016/zkl1-ppgo
dc.identifier.urihttp://hdl.handle.net/1903/26533
dc.language.isoenen_US
dc.subject.pqcontrolledPhysiologyen_US
dc.subject.pqcontrolledHealth sciencesen_US
dc.subject.pqcontrolledKinesiologyen_US
dc.subject.pquncontrolledcardiovascularen_US
dc.subject.pquncontrolledendothelialen_US
dc.subject.pquncontrolledil-6en_US
dc.subject.pquncontrolledinflammationen_US
dc.subject.pquncontrolledRace differencesen_US
dc.subject.pquncontrolledvascular functionen_US
dc.titleRACIAL DIFFERENCES IN VASCULAR FUNCTION FOLLOWING INDUCED ACUTE INFLAMMATIONen_US
dc.typeThesisen_US

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